Two cases of pseudoglaucoma were presented in this paper, which had been caused by lues and arteriosclerosis. We discussed the pathogenesis of pseudoglaucoma and reviewed the literature. Besides compression of the optic nerve and chiasm, ischemic optic neuropathy due to arteriosclerosis, lues, methanol intoxication, recurrent blood loss, and so forth, may. produce optic nerve excavation and visual field defect without increase of IOP. We had much difficulty in explaining Rheese¢¥s view of our case, in which ¢¥the optic canal was irregulary narrowed. We are of the opiaibn that narrowing of the optic canal may suggest secondary pathologic change due to chronic luetic optochiasmatic arachnoiditis.
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